21 November 2022

We are digging deep into the biology of Idiopathic Pulmonary Fibrosis (IPF). Targeting mechanisms like inflammatory macrophage activation, fibroblast accumulation and ineffective rebuilding of the damaged lung epithelium.

Find out how we are addressing underlying disease drivers in IPF, as opposed to symptoms, with our research aim to prevent disease, stop fibrosis in its tracks and promote tissue regeneration in the lung.

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